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Summary
DescriptionPotential Mechanisms of Cancer-Related Hypercoagulability.png |
English: Cancer-associated thrombosis can result from: (1) stasis, "i."e., direct pressure on blood vessels by, the——tumor mass, "poor performance status." And bed rest following surgical procedures; (2) iatrogenic, due——to treatment with antineoplastic medications; and (3) secretion of heparanase from malignant tumors that results in degradation of endogenous heparin. |
Date | |
Source | Potential Mechanisms of Cancer-Related Hypercoagulability. Nasser NJ, Fox J, Agbarya A. Cancers (Basel). 2020 Feb 29;12(3):566. https://doi.org/10.3390/cancers12030566 |
Author | Nicola J. Nasser, Jana Fox, and Abed Agbarya |
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Date/Time | Thumbnail | Dimensions | User | Comment | |
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current | 23:26, 19 February 2023 | 1,999 × 1,135 (794 KB) | Jul059 | Lossless file size reduction | |
06:24, 2 January 2021 | 1,999 × 1,135 (842 KB) | Sannecola | Uploaded a work by Nicola J. Nasser, Jana Fox, and Abed Agbarya from Potential Mechanisms of Cancer-Related Hypercoagulability. Nasser NJ, Fox J, Agbarya A. Cancers (Basel). 2020 Feb 29;12(3):566. https://doi.org/10.3390/cancers12030566 with UploadWizard |
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